Figure 1. An essential role for SWI/SNF ATPases in flower primordium initiation.

(A) ‘Pin’ inflorescence phenotype (white arrow) of brm syd double mutants. Scale bars = 1 mm. Allelic strength of mutants: syd-6, very weak; brm-3 weak; aMIRBRM, strong; syd-5 null. (B) Quantification of the flower primordia initiated in (A). n > 18. p-value: Mann–Whitney U test. (C) Enhancer tests using hypomorph mp-S319 mutant (Schlereth et al., 2010). White arrows point to ‘pin’ inflorescences. Scale bars = 1 mm. brm-1 null mutant (Hurtado et al., 2006) combined with the mp-S319 hypomorph mutant is seedling lethal like the mp-B4149 null mutant (Weijers et al., 2006) (Figure 1—figure supplement 1) and has developmental defects in the embryo (Figure 1—figure supplement 2). (D) Quantification of the flower primordia initiated in (C). n > 5. p-value: Mann–Whitney U test.

DOI: http://dx.doi.org/10.7554/eLife.09269.003

Figure 1—figure supplement 1. brm-1 null mutant mutants enhance mp-S319 hypomorph seedling phenotypes to phenocopy mp-B4149 null mutants.

Scale bars = 1 mm. Red arrows: number of cotyledons; white arrowhead: root growth arrest.

DOI: http://dx.doi.org/10.7554/eLife.09269.004

Figure 1—figure supplement 2. brm-1 null mutant enhance mp-S319 hypomorph embryo phenotypes.

Arrows: irregular cell division in hypophysis. Scale bars = 30 μm.

DOI: http://dx.doi.org/10.7554/eLife.09269.005