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. Author manuscript; available in PMC: 2016 Nov 1.
Published in final edited form as: Neurobiol Aging. 2015 Aug 15;36(11):2946–2953. doi: 10.1016/j.neurobiolaging.2015.08.008

Figure 2.

Figure 2

illustrates relationship of AD pathology, CAA and APOE genotypes. (A) and (B) show the distribution of the composite measure of AD pathology by CAA. The association of CAA with more burden of AD pathology is evident. (C) shows the percentages of moderate to severe CAA by APOE genotypes. The figure illustrates isoform-dependent pattern of APOE (i.e. ε4>ε2>ε3) in relation to CAA. (D) shows the percentages of pathologic AD diagnosis by APOE genotypes. Here, we observe a different pattern in relation to AD (i.e. ε4>ε3>ε2), as compared with CAA (C).