Table 2.
Changes of gastric mucosa at the edge of proximal gastric ulcers and gastric body one year after treatment, in relation to post-treatment H. pylori status, treatment regimens, ulcer relapse and ulcer location (n = 65)
|
Mucosal type change (%) |
||||||
|
Ulcer edge |
Gastric body |
|||||
| Improvementa(n = 11) | No changeb(n = 49) | Worseningc(n = 5) | Improvement(n = 5) | No change(n = 58) | Worsening(n = 2) | |
| H. pyloriinfection | ||||||
| Eradicated (n = 28) | 35.7d | 64.3 | 0 | 14.3 | 85.7 | 0 |
| Persistent (n = 37) | 2.7 | 83.8 | 13.5 | 2.7 | 91.9 | 5.4 |
| Ulcer location | ||||||
| Incisura (n = 52) | 15.4 | 76.9 | 7.7 | 5.8 | 90.4 | 3.8 |
| Body (n = 13) | 23.1 | 69.2 | 7.7 | 15.4 | 84.6 | 0 |
, Improvement, antral-type (A)→transitional type (T), A→body-type (B), or T→B;
, No change, A→A, T→T or B→B;
, Worsening, T→A, B→A or B→T.
, P < 0.001, compared with persistent infection.