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. 2015 Mar 16;14(8):1260–1267. doi: 10.1080/15384101.2015.1014150

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© 2015 The Author(s). Published with license by Taylor & Francis Group, LLC

© Eszter Pakai, Andras Garami, Tatiane B Nucci, Andrei I Ivanov, and Andrej A Romanovsky

This is an Open Access article distributed under the terms of the Creative Commons Attribution-Non-Commercial License (http://creativecommons.org/licenses/by-nc/3.0/), which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited. The moral rights of the named author(s) have been asserted.

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Figure 3. — Biochemical markers of renal disfunction in J/J and J/+ rats. (A) Plasma BUN levels do not differ between saline-treated J/J and J/+ rats and remain unchanged after administration of the low dose of LPS (10 μg/kg, iv). The high dose of LPS (1,000 μg/kg, iv) raises BUN in both genotypes, without any intergenotype difference. (B) Plasma creatinine levels do not differ between saline-treated J/J and J/+ rats and remain unchanged after administration of the low dose of LPS. The high dose of LPS raises plasma creatinine in both genotypes, without any intergenotype difference. Within each genotype, significant differences in the response to LPS (as compared to saline) are marked as ^## (P < 0.01) or ^### (P < 0.001).