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. 2015 Oct 21;15:252. doi: 10.1186/s12870-015-0614-2

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© Zhang et al. 2015

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 5 — Silencing of SlHUB1 resulted in increased resistance to P. syringae pv. tomato DC3000. Two-week-old seedlings were infiltrated with agrobacteria carrying pTRV2-SlHUB1, pTRV2-SlHUB2, pTRV2-SlMED21 or pTRV2-GUS conducts and disease assays were performed by vacuum infiltrating with Pst DC3000 at 4 weeks after VIGS infiltration. a Representative symptom of disease caused by Pst DC3000 at 4 dpi. b Bacterial growth in inoculated leaves of pTRV2-SlHUB1-, pTRV2-SlHUB2-, pTRV2-SlMED21- or pTRV2-GUS-infiltrated plants. Leaf samples were collected at 0 and 4 days after inoculation and bacterial growth was measured. Data presented are the means ± SD from three independent experiments and different letters above the columns indicate significant differences at p < 0.05 level