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. Author manuscript; available in PMC: 2016 Nov 13.
Published in final edited form as: Biochem Biophys Res Commun. 2015 Sep 30;467(2):407–412. doi: 10.1016/j.bbrc.2015.09.137

Figure 2.

Figure 2

Maternal diabetes induces Chk1 and Chk2 phosphorylation in embryos. A. Phosphorylation of Chk1 in E8.75 embryos from nondiabetic or diabetic dams, in the presence or absence of overexpression of SOD1. B. Phosphorylation of Chk2 in E8.75 embryos from nondiabetic or diabetic dams, in the presence or absence of overexpression of SOD1. Within each panel, the membrane was probed first for the phosphorylated protein and sequentially stripped for probing total protein and β-actin. The quantification of blot was showed in the bar graph. Experiments were repeated three times (n = 3). Values are the means ± SEM from three separate experiments. * indicates significant differences (P < 0.05) compared with other three groups.