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. 2015 Oct 22;10(10):e0140823. doi: 10.1371/journal.pone.0140823

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© 2015 Chen et al

This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are properly credited.

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Fig 1 — (A) Typical animal-tracking profiles within 3 min for the evaluation of neurological deficits. (B) Representative micro-PET analysis of brain function (glucose metabolism) in live mice at 24 h after stroke. Animal groups include sham, vehicle-treated animals (ischemic stroke, CIR), BHD-treated animals (CIR+BHD; 1.0 g/kg, p.o., twice daily), and TPA-treated animals (CIR+TPA; 10 mg/kg, i.v. at day one), with treatment administered 2 h after ischemic stroke. The experiment was repeated at least 3–5 times, with similar results. #p < 0.05 compared with the sham group; *p < 0.05 compared with the CIR group.