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. 2015 Oct 26;17:299. doi: 10.1186/s13075-015-0818-8

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© Jochmann et al. 2015

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 9 — Anti-CD68 antibody (ED1) immunoreactivity in lumbar dorsal root ganglia (DRGs) and its correlations with growth-associated protein 43 (GAP-43)–positive intraepidermal nerve fibres (IENFs) and behavioural data. Plot of the time course of GAP-43–positive IENFs in the left paw pad skin and ED1 immunoreactivity (macrophage invasion) in the lumbar DRGs (a). Correlation between ED1-immunoreactive cells in the DRGs and weight-bearing. Relative weight bearing for control animals was defined as 50 %, with equal weight on both legs. Control values are parenthesized (b). Correlation between ED1-immunoreactive cells in the DRGs and primary hyperalgesia. Control values in parentheses were set at 234.5 g, which was the average baseline value from animals before induction of AIA (SD was ±20.6 g) (c). Relationship of ED1 immunoreactivity and secondary hyperalgesia in the paw. Control values in parentheses were determined as the mean of baseline values from acute and chronic AIA animals before induction of AIA (27.9 g ± 7.6 g) (d)