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. 2015 Oct 28;35(43):14571–14584. doi: 10.1523/JNEUROSCI.1369-15.2015

Figure 1.

Figure 1.

Quiescence during DTQ and SIQ is mediated by different neuropeptides. A, Schematic representing the known pathways that control quiescence during DTQ and SIQ. In DTQ, the RIA neurons release NLP-22, whereas the RIS neuron releases an unidentified peptide. The aptf-1 gene is required for the function of the RIS neuron and the ceh-17 gene is required for the function of the ALA neuron. During SIQ, the ALA neuron releases the FLP-13 neuropeptides. It is unknown how these neuropeptides lead to behavioral quiescence. B, Feeding quiescence during SIQ does not require pathways that regulate DTQ. Mutants defective in RIA and RIS signaling (nlp-22 and aptf-1 mutants, respectively) have normal feeding quiescence during SIQ. Each point represents an observation from one worm and the horizontal bar represents the median of each group. n = 15 for each group. Statistical significance was calculated using the Mann–Whitney test. *p < 0.05.