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. 2015 Aug 10;66(21):6803–6817. doi: 10.1093/jxb/erv386

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© The Author 2015. Published by Oxford University Press on behalf of the Society for Experimental Biology.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/3.0/), which permits unrestricted reuse, distribution, and reproduction in any medium, provided the original work is properly cited.

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Fig. 8. — SNAC3 regulates stress responses in an ABA-independent manner. (A) No significant difference was observed between the SNAC3-OE, SNAC3-RNAi, and the control (ZH11) under ABA treatment. (B) Plant height of SNAC3-OE, SNAC3-RNAi, and ZH11 under ABA treatment. (C) Endogenous ABA content of SNAC3-OE and ZH11 WT leaves under normal conditions, drought stress, and heat stress conditions. Data represent the mean ±SE (n=3). (D) Expression analysis of SNAC3 in the ABA-deficient mutant phs3 under normal and drought stress conditions. Error bars indicate the SE based on three replicates.