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. 2015 Oct 30;16:885. doi: 10.1186/s12864-015-2095-y

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© Gely-Pernot et al. 2015

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 2 — Atrazine causes delayed meiosis in mice. a γH2AX staining changes during meiotic prophase I substages. b The distribution of meiotic substages in control (left panel) and ATZ-treated (right panel) mice; 600 cells from control and treated mice were classified according to γH2AX and SYCP3 staining. Note that the number of spermatocytes in the zygotene and early pachytene stages is increased in ATZ-treated animals, while the number of spermatocytes in the late stages decreased. c Surface spreads from control and ATZ-treated testis stained with anti-γH2AX (red) and anti-SYCP3 (green) antibodies. In control mice, the majority of meiotic cells have γH2AX marks at the sex bodies only. In ATZ-treated mice, the majority of meiotic cells have γH2AX marks at many chromosomes