Table 1.
The antiviral active components and their possible mechanisms for virus prevention.
Component | Antiviral mechanism | Viral type |
---|---|---|
GL | Affect release step while infectious HCV particles are infecting cells. | HCV |
Inhibit HCV full length viral particles and HCV core gene expression. | ||
Reduce adhesion force and stress between CCEC and PMN. | HSV | |
Block the degradation of nuclear factor κB inhibitor IκB. | CVB3 | |
Activate T lymphocyte proliferation. | DHV | |
Weaken H5N1-induced production of CXCL10, IL-6 and CCL5, and suppress H5N1-induced apoptosis. | H5N1 | |
Reduce HMGB1 binding to DNA, and inhibit influenza virus polymerase activity. | Influenza virus | |
Inactivate CVA16 directly, while the effect of anti-EV71 is associated with an event(s) during the virus cell entry. | CVA16 EV71 | |
Establish a resistance state to HSV1 replication. | HSV1 | |
GA | Reduce the levels of viral proteins VP2, VP6 and NSP2 at a step or steps subsequent to virus entry. | Rotavirus |
Prevent viral attachment, internalization and stimulate IFN secretion. | HRSV |