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. 2015 Jun 17;5(4):310–315. doi: 10.1016/j.apsb.2015.05.005

Table 1.

The antiviral active components and their possible mechanisms for virus prevention.

Component Antiviral mechanism Viral type
GL Affect release step while infectious HCV particles are infecting cells. HCV
Inhibit HCV full length viral particles and HCV core gene expression.
Reduce adhesion force and stress between CCEC and PMN. HSV
Block the degradation of nuclear factor κB inhibitor IκB. CVB3
Activate T lymphocyte proliferation. DHV
Weaken H5N1-induced production of CXCL10, IL-6 and CCL5, and suppress H5N1-induced apoptosis. H5N1
Reduce HMGB1 binding to DNA, and inhibit influenza virus polymerase activity. Influenza virus
Inactivate CVA16 directly, while the effect of anti-EV71 is associated with an event(s) during the virus cell entry. CVA16 EV71
Establish a resistance state to HSV1 replication. HSV1
GA Reduce the levels of viral proteins VP2, VP6 and NSP2 at a step or steps subsequent to virus entry. Rotavirus
Prevent viral attachment, internalization and stimulate IFN secretion. HRSV