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. 2015 Oct 28;8:3159–3167. doi: 10.2147/OTT.S93056

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© 2015 Luo et al. This work is published by Dove Medical Press Limited, and licensed under Creative Commons Attribution – Non Commercial (unported, v3.0) License

The full terms of the License are available at http://creativecommons.org/licenses/by-nc/3.0/. Non-commercial uses of the work are permitted without any further permission from Dove Medical Press Limited, provided the work is properly attributed.

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Loss of PTPRJ attenuates the inhibitory effects of knockdown miR-328 on migration and invasion of hepatocellular carcinoma (HCC) cells.

Notes: (A) At 48 hours posttransfection, Western blot analysis revealed that the expression level of PTPRJ protein in cells cotransfected with miR-328 inhibitor and si-PTPRJ was significantly lower than that in cells cotransfected with miR-328 inhibitor and si-con (*P<0.05). (B) Loss of PTPRJ could significantly attenuate the effects of miR-328 inhibitor on cell migration and invasion of both SMMC-7721 and BEL-7402 cells (*P<0.05).

Abbreviations: PTPRJ, protein tyrosine phosphatase, receptor type, J; si-con, matched control siRNA; miRNA, microRNA; si, small intefering.