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. 2015 Jun 24;35(11):1862–1870. doi: 10.1038/jcbfm.2015.145

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Copyright © 2015 International Society for Cerebral Blood Flow & Metabolism, Inc.

This work is licensed under a Creative Commons Attribution-NonCommercial-ShareAlike 4.0 International License. The images or other third party material in this article are included in the article's Creative Commons license, unless indicated otherwise in the credit line; if the material is not included under the Creative Commons license, users will need to obtain permission from the license holder to reproduce the material. To view a copy of this license, visit http://creativecommons.org/licenses/by-nc-sa/4.0/

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NeuN (A), Fluoro-Jade (B), and glial fibrillary acidic protein (GFAP; C) stained hippocampi from control, 1.5-, and 3-hour rats (shown in columns). In C, the top row shows the hippocampus at × 4 magnification with the identified inset boxes at × 10. (D) Shows tabulated NeuN counts from the hilus. For the NeuN counts, all kainate acid (KA) treated rats are significantly different from control, *P<0.05. For histology, all animals were killed immediately after the imaging study, 3 days after the status epilepticus period.