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. 2015 Nov 6;16:905. doi: 10.1186/s12864-015-2175-z

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© Larson et al. 2015

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 4 — The seasonal miR-132–mRNA regulatory network in HVC. a The relative expression changes of mRNA targets of miR-132 that were differentially regulated in at least one experimental group (i.e. LD + T 3D, 21, or LDW) compared to SD, as well as having an inverse correlation to miR-132 in the same comparison. A p-value of <0.005 and a fold change >1.5 at any time point were used as selection criteria for mRNAs presented in the heat map. b An interaction network of mRNAs that were differentially anti-expressed with a fold change > -1.5 and p < 0.005 from conditions in which miR-132 was also differentially expressed (i.e. fold change > 2.0). IPA network analyses revealed several key pathways were down-regulated during periods of HVC new neuronal addition and functional incorporation