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. Author manuscript; available in PMC: 2016 Nov 15.
Published in final edited form as: J Immunol. 2015 Oct 14;195(10):4685–4698. doi: 10.4049/jimmunol.1500806

Figure 6.

Figure 6

Protection against glomerulonephritis (GN) in Sle123→ApoA-Itg mice. (A) Periodic Acid Schiff (PAS) stained kidney sections showing representative glomeruli from the indicated 38 week old non-BMT control, control WT BMT and Sle123 BMT mice. (B) Average glomerular area (upper panel) and overall GN histological scores lower panel) in the indicated control (n=5), control BMT (n=5) and Sle123 BMT (n=6) groups (9 glomeruli scored per mouse) (‡p<0.05 by T test compared to indicated control, *p<0.05 by one way ANOVA within Sle123 BMT group). (C) Increases in urine albumin/creatinine ratio (ACR) in the indicated 38 week-old control (n=4), control BMT (n=4) and Sle123 BMT (n=6–8) animals (all data mean ± SD) (*p=0.052 by one way ANOVA, ‡p=0.049 compared to Sle123→WT by T test).