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. 2015 Apr 24;1(3):e1400203. doi: 10.1126/sciadv.1400203

Fig. 4. ABC-induced prolonged activation of homologous recombination–dependent repair in ATL cells.

Fig. 4

(A) Immunocytochemical staining with DAPI, anti-γH2AX, and anti-RAD51 recombinase. ED-40515(−) and Jurkat cells were treated with or without 100 μM ABC for 2 days. The percentages of γH2AX and RAD51 focus–positive cells, defined as cells containing more than five foci per cell, are shown. (B) Immunocytochemical staining with DAPI, anti-γH2AX, and anti-RAD51. ED-40515(−) and Jurkat cells were treated with or without 100 μM ABC for 2 days. The merged images display frequent colocalization of γH2AX and RAD51.