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. 2015 Nov 10;16:75. doi: 10.1186/s12868-015-0213-z

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© Papatheodoropoulos. 2015

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 5 — Dependence of spfps_-nmda on NMDARs and glutamate release. Example recordings illustrating that spfps_-nmda were completely abolished by the competitive antagonist of NMDARs CPP (a) or the open channel blocker of the same receptors MK-801 (b). Spfps_-nmda were also blocked by activation of adenosine A₁ receptors by their agonist 2-chloroadenosine (c). Data shown in “b” and “c” were obtained from two DH slices