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. Author manuscript; available in PMC: 2015 Nov 11.
Published in final edited form as: Eur J Neurosci. 2014 Apr 21;40(2):2359–2377. doi: 10.1111/ejn.12592

Fig. 1.

Fig. 1

(A) Drawings of the largest (gray) and smallest (black) accepted lesions of amygdala central nucleus (CeA) and lateral hypothalamus (LH), based on Nissl-stained sections, in experiment 1. All lesions are shown in the same hemisphere, regardless of whether they were made in the right or left hemisphere, and whether the CeA and LH lesions were made ipsilaterally or contralaterally. (B–E) Sample Nissl-stained sections of lesioned (B and D; lesions indicated by dotted borders) and unlesioned (C and E) CeA (B and C) and LH (D and E). The sections all come from a single rat, which received contralateral lesions of CeA and LH. Images in (C and D) were reversed left-to-right to portray all sections in a common orientation, matching (A). BLA, basolateral amygdala; DMH, dorsomedial hypothalamus; fx, fornix; IC, intercalated nuclei; mtt, mammillothalamic tract; vlt, ventrolateral hypothalamic tract. Drawings of brain sections in (A) are from Swanson (1998); used by permission of Elsevier.