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. Author manuscript; available in PMC: 2016 Dec 1.
Published in final edited form as: Hypertension. 2015 Oct 5;66(6):1227–1239. doi: 10.1161/HYPERTENSIONAHA.115.06370

Figure 2.

Figure 2

LTB4 promotes proliferation, migration and differentiation in HPAAF. Proliferation of HPAAF with increasing doses of LTB4 treatment for 72hrs was measured by A, the MTT assay, B, cell counting, C, BrdU assay. Data are presented as mean ± SEM. (*: p<0.05) D, PCNA expression was measured by western blot after treatment with LTB4 for 24hr. E, F, Migration of HPAAF after LTB4 exposure were determined and quantified by Boyden Chamber assay. Data are presented as mean ± SEM. (*: p<0.05) G, H α-SMA and p-p38 MAPK expression after LTB4 treatment for 24hr as determined by western blot. β-actin was used as a loading control. The experiments were repeated three times.

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