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. 2015 Jul 16;6(7):e1818. doi: 10.1038/cddis.2015.182

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Copyright © 2015 Macmillan Publishers Limited

Cell Death and Disease is an open-access journal published by Nature Publishing Group. This work is licensed under a Creative Commons Attribution 4.0 International License. The images or other third party material in this article are included in the article's Creative Commons license, unless indicated otherwise in the credit line; if the material is not included under the Creative Commons license, users will need to obtain permission from the license holder to reproduce the material. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/

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Aip1 deletion in either germ cells or Sertoli cells caused alterations in E-cadherin distribution pattern in both germ cells and Sertoli cells. (a and b) Co-immunostaining of E-cadherin (red) and PLZF (green) of testis sections from the P7 control, geAip1^−/− (a) and seAip1^−/− (b) mice. (c and d) Co-immunostaining of E-cadherin (red) and SOX9 (Sertoli cell marker, green) of testis sections from the P7 control, geAip1^−/− (c) and seAip1^−/− (d) mice. White arrows point to regions of Sertoli cells where E-cadherin level is upregulated, and the asterisk (*) signs mark germ cells whose E-cadherin distribution pattern is affected. Cell nuclei were stained with DAPI (blue). Scale bars: 10 μm