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. 2015 Nov 1;4(1):417–424. doi: 10.1089/biores.2015.0006

Table 2.

FACS Analysis on BMA and cBMA from the MarrowStim P.A.D. Kit (n = 6)

CD marker Function and cell type BMA (%) cBMA (%)
R1 gate Lymphocytes 17.4 ± 6.6 20.5 ± 3.5
R2 gate Monocytes 4.0 ± 1.2 4.3 ± 1.5
R3 gate Granulocytes 75.5 ± 5.8 71.2 ± 7.2
VEGFR-2 Tyrosine kinase receptor (endothelial cells and precursors) 5.6 ± 1.8 7.5 ± 3.2
CD3 Antigen receptor (T cells) 9.0 ± 3.3 11.5 ± 2.9
CD14 Surface glycoprotein (monocytes and macrophages) 5.4 ± 2.5 4.8 ± 3.3
CD34 Transmembrane glycoprotein (hematopoietic and endothelial progenitor cells) 0.9 ± 1.0 1.0 ± 0.6
CD45 Transmembrane protein tyrosine kinase (white blood cells and hematopoietic stem cells) 91.1 ± 4.9 90.7 ± 6.6
CD56 NCAM-1 (heparin-binding glycoprotein; T cells) 8.3 ± 8.5 7.7 ± 5.8
CD105 TGF-β receptor, endoglin (SH2; endothelial cells, monocytes, macrophages, a subpopulation of hematopoietic stem cells, and cultured mesenchymal stem cells) 4.1 ± 3.4 9.3 ± 16.1
CD117 Tyrosine kinase receptor (hematopoietic stem cells, B cells, and T cells) 3.2 ± 5.5 1.8 ± 1.2
CD133 Trasmembrane protein (hematopoietic stem cells and endothelial cells) 0.2 ± 0.1 0.2 ± 0.1
Stro-1 Bone marrow stromal cells 0.8 ± 0.6 1.1 ± 0.6

There were no significant differences between the BMA and the cBMA for all markers analyzed, except for the VEGFR-2 receptor (p = 0.04).

FACS, fluorescence-activated cell sorting; TGF-β, transforming growth factor-β.