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. 2015 Nov 19;8:76. doi: 10.1186/s13041-015-0169-z

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© Liu et al. 2015

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 1 — Intracolonic injection with zymosan induced visceral pain-like behaviors and enhanced expression of AMPA receptor in the ACC in mice. a The time course for injected with zymosan and behavioral test. b-d Mice injected with zymosan exhibited obvious visceral pain behavior (b), and reduced travel distance (c) and vertical counts (d) on day 1, 7, and 14 as compared with control. e Representative Western blot for GluA1 and GluA2/3 in the ACC obtained from control and zymosan-injected mice. f-g The expressions of GluA1 (f) and GluA2/3 (g) were significantly enhanced on day 1, 7, and 14 in the ACC of mice injected with zymosan as compared to those of control mice. N = 6 mice per group. * P < 0.05, ** P < 0.01 vs. control