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Staphylococcus aureus signaling mechanism after contact and internalization by osteoblasts. After internalization, Staphylococcus aureus can escape from vesicle, be disrupted from inside the vesicle or persist inside osteoblasts through a SCV phenotype. Staphylococcus aureus can also interact with extracellular receptors TLR2 and TNFR-1 and with intracellular receptors TLR9 and NODs after its internalization into osteoblasts thanks to α5β1 integrin and actin filaments.
