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. 2015 Sep 17;10(5):1675–1680. doi: 10.3892/etm.2015.2752

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Copyright: © Zhou et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 2. — Immunofluorescence staining of LC3-II in the bone tissue near the fracture site. (A) In the control group, LC3-II-positive cells were sparse; in the fracture group, the population of LC3-II-positive cells increased after 6 h, peaked at 24 h and then gradually decreased at 3 days. (B) Analysis of the percentage of cells in fracture-site tissue with punctuate LC3-II fluorescence. Results are presented as the mean ± standard deviation. *P<0.05 vs. control. Scale bar, 100 µm; magnification, ×400. LC3-II, microtubule-associated protein II light chain 3.