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. 2015 Dec 1;17:106. doi: 10.1186/s12968-015-0197-y

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© Thomas et al. 2015

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 4 — Baseline and 3d post-infarct CMR with gadolinium-DTPA. a. Baseline myocardial perfusion territory images (seen as white enhancement) of the mid LAD from apex to base. This territory represented 39 % of the overall left ventricular mass. b. LGE images showing regions of persistent MVO (red arrow) and infarct tissue (yellow arrow). Infarct size as a percent of total left ventricular mass (center) was significantly increased in DCE animals compared to controls (*p = 0.04) (Control n = 15; DCE n = 13). The DCE group also demonstrated an increased infarct size relative to the baseline perfusion territory at-risk (p = 0.08). c. EGE images depicting MVO (Left, red arrow). A non-significant increase in the proportion of animals displaying MVO (center) and persistent MVO (right) was observed in the DCE group