Figure 6. Activation markers of histone modification (H3AcK9 & H3K4me2) in the γ-globin gene promoter in HbE β-thalassemia progenitors treated with Benserazide (BEN).
H3K4me2 and H3AcK9 enrichment in the γ-globin gene promoter is shown in Benserazide-treated HbE β-thalassemia progenitors and vehicle-treated control. The error bars represent the SEM. Asterisks indicate significant differences between the vehicle controls and Benserazide-treated cells (*p<0.05).