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. Author manuscript; available in PMC: 2016 Dec 15.
Published in final edited form as: Cancer. 2015 Sep 15;121(24):4348–4358. doi: 10.1002/cncr.29644

Figure 3. TFF1 attenuates H. pylori-induced activation of NFκB signaling through dephosphorylation of IKKα/β and NFκB-p65 proteins.

Figure 3

A) Western blot analysis of the indicated proteins showed that the reconstitution of TFF1 decreases protein levels of p-IKKα/β and pNFκB-p65 after H. pylori infection. Protein loading was normalized for equal levels of β-actin and the infection with H. pylori was confirmed using CagA specific antibody. B–C) The relative density of p-IKK a/β (B) and p-NFκB-p65 (C) normalized to β-Actin are presented as graphs produced from analysis of three independent blots by ImageJ software. The results are expressed as mean ± SEM of at least 3 independent experiments.