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. 2015 Dec 10;6:257. doi: 10.3389/fneur.2015.00257

Table 2.

Potential remyelinating and neuroprotective therapies in multiple sclerosis.

Drug Proposed mechanism Results Reference
Anti-ASIC-1 Blockage of ASIC-1 prevents excessive intracellular accumulation of injurious Na(+) and Ca(2 +) in MS lesions Clinical studies suggest neuroprotection as measured by brain atrophy during treatment compared with pretreatment. (105, 106)
Anti-LINGO-1 Function-blocking anti-LINGO-1 antibodies enhance OPC differentiation and myelination Phase 2 trial in patients with a first episode of optic neuritis showed an improvement on nerve impulse conduction along the affected optic nerve. Phase 2 trial in RRMS is ongoing. (107, 108)
Benztropine Antagonism of M1/M3 muscarinic acetylcholine receptors with subsequent stimulation of oligodendrocyte differentiation In experimental models of MS, benztropine induced the differentiation of OPCs, and enhanced remyelination. (109)
Guanabenz α2 adrenergic receptor agonist. Protects oligodendrocytes by preventing dephosphorylation of eIF2, increasing oligodendrocyte survival and prevention of myelin loss. Preclinical studies demonstrated improvement of deficits in EAE. Phase I clinical studies are ongoing. (110, 111)
Laquinimod Modified quinolone derivative; reduces microglia and astrocyte activation; increases neuroprotection and myelin preservation Clinical studies suggest neuroprotection as measured by brain atrophy in treated versus untreated patients. (112117)
Miconazole and clobetasol Activates eIF2, TX/RXR, and cholesterol signaling Promoted oligodendrocyte differentiation and enhanced remyelination in in vivo models (118)
Olesoxime Decreases oxidative stress. Promotes oligodendrocyte maturation and myelin synthesis Accelerated oligodendrocyte maturation and enhanced myelination in vitro and in vivo without affecting oligodendrocyte survival or proliferation. Phase 1 trail in MS patients completed. (119, 120)
Quetiapine fumarate Stimulates proliferation and maturation of oligodendrocytes, increases neurotrophic factors, and inhibits activated microglia, astrocytes, and T lymphocytes Remyelinating and neuroprotective properties in EAE (121, 122)
rHIgM22 rHIgM22 binds to the surface of oligodendrocytes promoting myelin repair Preclinical studies indicate that it may protect oligodendrocytes and stimulate myelin repair. Phase I study demonstrated acceptable safety profile. (123125)

OPC, oligodendrocyte precursor cells; eIF2, Eukaryotic Initiation Factor 2; CNS, Central Nervous System; LINGO, leucine-rich repeat and immunoglobulin-like domain-containing, Nogo receptor-interacting protein; rHIgM22, recombinant human IgM antibody 22; EAE, experimental autoimmune encephalomyelitis; ASIC-1, acid-sensing (proton gated) ion channel 1.