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. 2015 Dec 12;8:86. doi: 10.1186/s13041-015-0175-1

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© Xu et al. 2015

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 1 — Induction of CTA by a rotatory stimulus or intra-vestibular nuclei microinjection of AVP and the effects of the V1bR antagonist (n = 10). a, indication of the microinjection area in the VN (Nissl staining). b, induction of CTA by AVP microinjection into the VN and the blocking effect of V1bR antagonist SSR149415. c, induction of CTA after rotatory stimulus and the blocking effect of SSR149415 microinjection into the VN. d, induction of CTA after AVP intraperitoneal injection (i.p.) and the blocking effect of SSR149415 microinjection into the VN. ^* P < 0.05, ^** P < 0.01, vs. vehicle; ^# P < 0.05, ^## P < 0.01 vs. AVP 10 ng or 30 ng groups, respectively; ^Δ P < 0.05, vs. AVP (i.p.) used alone