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. 2015 Dec 6;6:248. doi: 10.1186/s13287-015-0237-4

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© Perea-Gil et al. 2015

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 3 — Engraftment of a decellularized myocardial ECM scaffold embedded with cells in a swine myocardial infarction model. a Lyophilized and gamma ultraviolet sterilized decellularized myocardial ECM scaffold. b, c Decellularized scaffold after the addition of peptide hydrogel (b) and porcine adipose tissue-derived progenitor cells (c). Scale bars = 1 cm. d Image of the myocardial infarction site, induced by double ligation in the first marginal branch of the circumflex artery (indicated with white arrow). e Reseeded decellularized scaffold placed over the injured myocardium. Scaffold is indicated with yellow dotted lines. f Presence of the implanted scaffold on the infarcted area in explanted hearts 28 days after sacrifice. The remaining scaffold is highlighted with yellow dotted lines