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. 2015 Dec 18;15:283. doi: 10.1186/s12862-015-0558-z

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© Fellner et al. 2015

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 3 — Mutations introduced to arrest the translation of nog1 (left) or citC (right) strand specifically. The numbers indicate the distance of the sequence shown from the start codons of nog1 and citC respectively. Note that the amino acid sequence of the reading frame in antisense remains unchanged in both cases. The positions of the mutations are shown in red