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. 2015 Dec 22;11(12):e1005711. doi: 10.1371/journal.pgen.1005711

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© 2015 Bennett et al

This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are properly credited.

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Fig 8 — CD68+ cells but not smooth muscle actin cells differ among strains comprising the HMDP. Representative results from immunostained lesions for sm-α actin are shown in Panels A-E and CD68 in panels. C57BL/6J (A,F), A/J (B,G), BALB/cJ (C, H), C3H/HeJ (D, I), DBA/2J (E, J). (K, L) Impact of genetic background on lesion morphology in C57BL/6, A/J, C3H/HeJ, BALB/c and DBA/2, 5 progenitor strains for the HMDP recombinant inbred strains. Immunohistological staining for macrophages (CD68) (K) or smooth muscle cells (smooth muscle α-actin) (L) was measured as percent of total atherosclerotic lesion area ± SEM.