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. 2015 Dec 23;10(12):e0145666. doi: 10.1371/journal.pone.0145666

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© 2015 Ma et al

This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are properly credited.

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Fig 2 — Histological staining with PAS and hematoxylin shows the kidney structure of (A) a normal mouse. In comparison, there is severe damage to glomeruli (capillary thrombosis, glomerulosclerosis, crescent formation) at day 15 of glomerulonephritis in (B) a mouse receiving no treatment and (C) a mouse receiving vehicle, which is attenuated in (D) a mouse treated with BR-4628. Graphs show (E) the severity score of glomerular tuft damage (0–4+) and (F) the percentage of glomeruli with crescents in kidneys from normal control (NC) mice and mice with glomerulonephritis (GN) receiving no treatment (No Tx), vehicle or BR-4628. Magnification: (A-D) x400. Graph Data = mean ± SEM; n = 8. *p<0.05, ***p<0.001, NS = not significant.