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. Author manuscript; available in PMC: 2016 Dec 1.
Published in final edited form as: Alcohol. 2015 Oct 27;49(8):781–794. doi: 10.1016/j.alcohol.2015.08.006

Fig. 4. Ethanol potentiation is prevented by a CB1 antagonist/inverse agonist in vibrodissociated BLA projection neurons.

Fig. 4

A, B) Representative traces obtained from single neurons before and during ethanol application using fast perfusion in the absence and presence of the CB1 antagonist SR (1 μM). (scale bar 200 pA, 60 sec). C) Time course of the effect of 80-mM ethanol on sIPSC frequency in the presence and absence of the CB1 antagonist SR (n = 12 cells). D) Bar graph showing the average ethanol effect on sIPSC amplitude and frequency in the presence and absence of the CB1 antagonist/inverse agonist (*p < 0.05 vs. baseline, paired t test).