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. 2015 Jul 16;6(28):25828–25842. doi: 10.18632/oncotarget.4558

Figure 7. α-Mangostin and rTRAIL induced significant growth suppression of CSC-1 and -2 cells by up-regulating expression of DR5.

Figure 7

Upper bar graphs: Breast cancer stem-like CSC-1 and CSC-2 cells were treated with α-mangostin (2, 5, 7 μM) and/or rTRAIL (7 ng/ml) for 48 h. The cell viability was estimated at 48 h after the treatment. Data were obtained from 3 independent experiments. The cell viability of the control (0; DMSO alone) is indicated as 100%. Lower blot: Western blot analysis was performed to determine the expression of DR5 and activation of caspase-8, with β-actin as the internal control.