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. 2015 Jun 30;6(28):26119–26128. doi: 10.18632/oncotarget.4444

Figure 1. Inhibition of cell glucose metabolism by FF in the human glioblastoma cells.

Figure 1

A. Cells were treated with various concentrations (25, 50 and 100 μM) of FF. After 48 h, ECAR was measured by the Glycolysis Stress tests. B. GW9662 (10 μM), a PPAR inhibitor, restores FF-induced the inhibition of cell glucose metabolism. *P < 0.05, **P < 0.01. Results are representative of at least three independent experiments.