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. 2015 Oct 23;7(4):2124–2133. doi: 10.3390/cancers7040881

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© 2015 by the authors; licensee MDPI, Basel, Switzerland.

This article is an open access article distributed under the terms and conditions of the Creative Commons by Attribution (CC-BY) license (http://creativecommons.org/licenses/by/4.0/).

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Effect of p38δ MAPK status on oesophageal cancer cell growth and migration. Oesophageal p38δ MAPK negative (KE-3 and -8, KYSE-70, OE-21, and OC-1) (A) and positive (KE-4, -5, -6, and -10, KYSE-450, OC-3, OE-19, -21 and -33) (B) cell lines were examined for their cell proliferation rate. Cells were seeded (3 × 10⁴) in six-well plates and counted for 24–120 h (A,B); (C) Effect of p38δ on cell migration using a Boyden Chamber as described in the Experimental Section. The results shown are mean ± S.E. of three independent experiments.

Effect of p38δ MAPK status on oesophageal cancer cell growth and migration. Oesophageal p38δ MAPK negative (KE-3 and -8, KYSE-70, OE-21, and OC-1) (A) and positive (KE-4, -5, -6, and -10, KYSE-450, OC-3, OE-19, -21 and -33) (B) cell lines were examined for their cell proliferation rate. Cells were seeded (3 × 10⁴) in six-well plates and counted for 24–120 h (A,B); (C) Effect of p38δ on cell migration using a Boyden Chamber as described in the Experimental Section. The results shown are mean ± S.E. of three independent experiments.