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CBK mice develop a defective inflammation-resolution axis. mRNA expression of Alox-5, Hmox-1, ptgs-2 (A) and Alox-12, Alox-15 (B). C: immunoblot representing 5-LOX, COX-2, and HO-1 in control and CBK mice at 28 wk of age. D: densitometry analyses of 5-LOX, COX-2, and HO-1. β-Actin was used as loading control. *P < 0.05 vs. age-matched control; n = 5 mice/group.
