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. 2015 Dec 15;29(24):2603–2616. doi: 10.1101/gad.267633.115

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© 2015 Kadri et al.; Published by Cold Spring Harbor Laboratory Press

This article is distributed exclusively by Cold Spring Harbor Laboratory Press for the first six months after the full-issue publication date (see http://genesdev.cshlp.org/site/misc/terms.xhtml). After six months, it is available under a Creative Commons License (Attribution-NonCommercial 4.0 International), as described at http://creativecommons.org/licenses/by-nc/4.0/.

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Figure 6. — Defective erythroid maturation in Gata1^S310A mice treated with PPP. (A) Comparison of spleens from Gata1 and Gata1^S310A mice after 20 d of PPP treatment (left) and BFU-E colony assays of these spleens in M3234 methylcellulose medium (right). n = 4. (B) Perl's Prussian blue staining of paraffin sections of these spleens. Bar, 200 μm. (C,D) Ter119 (X-axis) and CD71 (Y-axis) FACS analysis of the Sytox-negative living cell population of the spleen (C) and bone marrow (D) from Gata1 and Gata1^S310A mice. Numbers show the percentage of cells within a particular region.