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. 2015 Sep 8;44(1):117–133. doi: 10.1093/nar/gkv885

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© The Author(s) 2015. Published by Oxford University Press on behalf of Nucleic Acids Research.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by-nc/4.0/), which permits non-commercial re-use, distribution, and reproduction in any medium, provided the original work is properly cited. For commercial re-use, please contact journals.permissions@oup.com

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Figure 3. — Elevated EPC1 expression in invasive and chemoresistant tumor cells. (A) Endogenous EPC1 and E2F1 expression levels in different cancer cell lines as assessed by Western blot. Actin was used for loading control. (B) Protein levels of endogenous E2F1 and EPC1 in epirubicin-resistant MCF7 human breast cancer cells (MCF7-EPIR) and parental cells (left). Protein levels of E2F1 and EPC1 in cisplatin (cDDP, 30 μM) treated SK-Mel-147 cells expressing sh.E2F1 or sh.control compared with untreated cells (right). Actin was used for loading control.