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. 2016 Jan 8;14:6. doi: 10.1186/s12967-015-0752-1

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© Unger et al. 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 9 — Changes in isoform phosphorylation of the target signaling protein, c-MET, in response to ischemia. An overview of ischemia-dependent regulation of isoforms of c-MET in normal tissue of all patients analyzed is shown in a. Corresponding data for tumor tissue are shown in b. Results were displayed as fold changes normalized to sample NC5/N0 and in tumor tissue to sample NC5/T0. Kruskal–Wallis test and Dunn test for multiple comparisons were used for statistical analysis. N normal tissue, T tumor tissue, 0 before surgery, 10, 20, 45 10, 20, 45 min after resection; time points (10, 20, 45) are compared with time point 0; ^a p ≤ 0.05