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. 2015 Dec 30;33(4):328–336. doi: 10.3857/roj.2015.33.4.328

Fig. 4. S-allylcysteine (SAC) inhibited phosphorylation of Met in the Met-focal adhesion kinase (FAK) signaling pathway stimulated by hepatocyte growth factor. HNE1 showed direct phosphorylation between Met and FAK while HONE1 presented indirect phosphorylation via Src. HGF, hepatocyte growth factor; MMP, matrix metalloproteinase.

Fig. 4