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. 2015 Dec;110(8):996–1002. doi: 10.1590/0074-02760150184

Fig. 1. : biological effects of the Trypanosoma cruzi strain Berenice-62 infection in H9c2 cellular survival. Cells were grown and infected with the parasite and the initial 48 h after parasitic infection were investigated. A: infected cells were analysedvia fluorescence microscopy; the nuclear material was labelled with DAPI (Bar = 50 μm and 100 μm). The representative rates of infected and uninfected cells are demonstrated in the circle; B: cellular viability assays after T. cruzi cellular infection; C: cellular and molecular analyses of the effect of the PTEN inhibitor (SF1670) in H9c2 cells. Cellular viability was measured by the formazan production and plotted in graphs. The western blot analyses demonstrated the effect of the drug in AKT phosphorylation. The presented values are the average from three independent experiments and the error bars represent the standard deviation of the mean. One-way analysis of variance testing showed significant differences between the control and cell samples and the significance level was set at p < 0.05 (***).

Fig. 1