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. 2016 Jan 11;12(1):e1005383. doi: 10.1371/journal.ppat.1005383

Fig 10. Ability of EBNA3C to bind to RBPJ appears to be essential for repression of ADAM28, ADAMDEC1 and COBLL1.

Fig 10

(A-C) Infection of primary B cell with either wild type (wtA5), EBNA3C knockout (3CKO), EBNA3C revertant (3CRev) or RBPJ binding mutant EBNA3C recombinant EBV. Gene expression of ADAM28 (A), ADAMDEC1 (B) and COBLL1 (C) was normalised to GNB2L1 and is shown relative to uninfected primary B cells.