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. 2016 Jan 12;9:71. doi: 10.3389/fnint.2015.00071

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Copyright © 2016 Zakirova, Crynen, Hassan, Abdullah, Horne, Mathura, Crawford and Ait-Ghezala.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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No alterations in cell morphology detected 22.5 months post exposure to PB+PER. Nissl staining revealed no gross morphological changes in nuclei/cell body of pyramidal neurons post exposure to PB+PER (A–D). Similarly, the majority of cells in the hippocampi and cerebral cortices of PB+PER exposed mice (F,H) as compared to controls (E,G) were free from damaged and swollen axons and degenerated neurons when compared to a positive control (PSAPP mouse model of Alzheimer's Disease; see inset in E). Representative images were taken at 40X magnification (scale bar represents 20 μm).