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. 2015 Nov 24;147(2):178–189. doi: 10.1111/imm.12549

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Lymphocyte and monocyte function in colon and blood. (a) Dendrimer glucosamine (DG; 334 mg) did not change the % CD3⁺ CD4⁺ lymphocytes or % CD3⁺ CD8⁺ lymphocytes in colon but reduced their Ki67 activation on day 6. (b, c) DG (334 mg) had no effect on the % tumour necrosis factor‐α (TNF‐α) ‐producing CD3⁺ CD4⁺ lymphocytes and CD3⁺ CD8⁺ lymphocytes or on the % TNF‐α, interleukin‐1β (IL‐1β), IL‐6, IL‐8‐producing CD14⁺ monocytes in blood after in vitro stimulation with PMA/ionomycin/Brefeldin‐A (days 0 and 6). Normal (■/□); Shigella (); DG 214 mg (); DG 334 mg(). Mean ± SEM with two‐tailed Mann–Whitney test (*P < 0·05).

Inline graphic — Lymphocyte and monocyte function in colon and blood. (a) Dendrimer glucosamine (DG; 334 mg) did not change the % CD3⁺ CD4⁺ lymphocytes or % CD3⁺ CD8⁺ lymphocytes in colon but reduced their Ki67 activation on day 6. (b, c) DG (334 mg) had no effect on the % tumour necrosis factor‐α (TNF‐α) ‐producing CD3⁺ CD4⁺ lymphocytes and CD3⁺ CD8⁺ lymphocytes or on the % TNF‐α, interleukin‐1β (IL‐1β), IL‐6, IL‐8‐producing CD14⁺ monocytes in blood after in vitro stimulation with PMA/ionomycin/Brefeldin‐A (days 0 and 6). Normal (■/□); Shigella (); DG 214 mg (); DG 334 mg(). Mean ± SEM with two‐tailed Mann–Whitney test (*P < 0·05).