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. 2015 Dec 9;172(24):5744–5869. doi: 10.1111/bph.13348
Nomenclature GPR152 GPR153 GPR160 GPR161 GPR162
HGNC, UniProt GPR152, Q8TDT2 GPR153, Q6NV75 GPR160, Q9UJ42 GPR161, Q8N6U8 GPR162, Q16538
Comments A C‐terminal truncation (deletion) mutation in Gpr161 causes congenital cataracts and neural tube defects in the vacuolated lens (vl) mouse mutant [1226]. The mutated receptor is associated with cataract, spina bifida and white belly spot phenotypes in mice [994]. Gene disruption is associated with a failure of asymmetric embryonic development in zebrafish [1085].