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. 2016 Jan 15;30(2):233–247. doi: 10.1101/gad.263327.115

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© 2016 Takano et al.; Published by Cold Spring Harbor Laboratory Press

This article is distributed exclusively by Cold Spring Harbor Laboratory Press for the first six months after the full-issue publication date (see http://genesdev.cshlp.org/site/misc/terms.xhtml). After six months, it is available under a Creative Commons License (Attribution-NonCommercial 4.0 International), as described at http://creativecommons.org/licenses/by-nc/4.0/.

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Figure 5. — Suppression of Prrx1a/b attenuates pancreatic cancer formation, hematogenous dissemination, and metastatic colonization. (A) Experimental design for orthotopic transplantation using KPC2 control and Prrx1a/b knockdown cells. (B) Primary tumor volumes of KPC2 control and shPrrx1a/b groups. (C) Box plot histograms indicate the number of CTCs between control and Prrx1a/b knockdown cells. (D) Representative pictures of liver metastases and the frequency of metastases in two groups. Mean ± SEM is indicated below the box plot histograms. Bar, 100 μm.