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. 2015 Dec 1;8(12):1531–1542. doi: 10.1242/dmm.022426

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© 2015. Published by The Company of Biologists Ltd

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/3.0), which permits unrestricted use, distribution and reproduction in any medium provided that the original work is properly attributed.

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Fig. 1. — Phenotype of p53-knockout mice. (A) View of the palate of an E18.5 p53 homozygote embryo (p53/p53) and a control sibling (p53/+) embryo. The double-headed arrow indicates the cleft palate. (B) Photograph of a p53/p53 E18.5 embryo with the eyelids open (arrow) phenotype. (C) Sections through the lungs of an E18.5 p53 homozygote embryo (p53/p53) and a control sibling (p53/+) embryo, showing the homozygous lung phenotype. Note the reduced number and width of airspaces in the affected p53/p53 homozygote (see also Fig. 2). Scale bars: 200 μm.